[PDF][PDF] COX inhibitors and thromboregulation
AJ Marcus, DJ Pinsky - The New England journal of medicine, 2002 - med.stanford.edu
AJ Marcus, DJ Pinsky
The New England journal of medicine, 2002•med.stanford.eduROM a historical perspective, there is perhaps no more interesting therapeutic saga than
that of aspirin, which began as a folk remedy, distilled from willow bark, and became a
lifesaving preventive treatment for ischemic cardiovascular disease. Aspirin primarily inhibits
the cyclooxygenase (COX)-dependent synthesis of eicosanoids, which are the end products
of metabolism of essential fatty acids and include prostacyclin and thromboxane A2.
Numerous studies have
that of aspirin, which began as a folk remedy, distilled from willow bark, and became a
lifesaving preventive treatment for ischemic cardiovascular disease. Aspirin primarily inhibits
the cyclooxygenase (COX)-dependent synthesis of eicosanoids, which are the end products
of metabolism of essential fatty acids and include prostacyclin and thromboxane A2.
Numerous studies have
ROM a historical perspective, there is perhaps no more interesting therapeutic saga than that of aspirin, which began as a folk remedy, distilled from willow bark, and became a lifesaving preventive treatment for ischemic cardiovascular disease. Aspirin primarily inhibits the cyclooxygenase (COX)-dependent synthesis of eicosanoids, which are the end products of metabolism of essential fatty acids and include prostacyclin and thromboxane A2. Numerous studies have
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